P02-014 - Consequences of Arginine 92 mutations in TNFR1

نویسندگان

  • I Jéru
  • S Charmion
  • E Cochet
  • B Copin
  • G Le Borgne
  • P Cathebras
  • J Gaillat
  • P Duquesnoy
  • S Karabina
  • V Hentgen
  • S Amselem
چکیده

Introduction TNFRSF1A is involved in a Mendelian autosomal dominant autoinflammatory disorder called TNFR-associated periodic syndrome (TRAPS). Most TNFRSF1A mutations are missense changes and, apart from those affecting conserved cysteines, their deleterious effect remains often questionable. This is especially true for the frequent R92Q mutation, which might not be responsible for TRAPS per se but represents a susceptibility factor to multifactorial inflammatory disorders.

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Involvement of the Same TNFR1 Residue in Mendelian and Multifactorial Inflammatory Disorders

OBJECTIVES TNFRSF1A is involved in an autosomal dominant autoinflammatory disorder called TNFR-associated periodic syndrome (TRAPS). Most TNFRSF1A mutations are missense changes and, apart from those affecting conserved cysteines, their deleterious effect remains often questionable. This is especially true for the frequent R92Q mutation, which might not be responsible for TRAPS per se but repre...

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عنوان ژورنال:

دوره 11  شماره 

صفحات  -

تاریخ انتشار 2013